By Edward C. Feldman DVM DACVIM, Richard W. Nelson DVM, Visit Amazon's Claudia Reusch Page, search results, Learn about Author Central, Claudia Reusch, , J. Catharine Scott-Moncrieff
No different name bargains such commitment to the intensity, adventure, and concentration of endocrinology as Canine and tom cat Endocrinology, 4th Edition. complete assurance contains almost each universal and unusual situation in endocrinology, plus the main up to date info on food, geriatric care, pathophysiology, checking out techniques, and low-budget and expedient diagnostic protocols. With its logical, step by step counsel for choice making, prognosis, and prescribing, you'll be well-equipped to deal with the vast spectrum of endocrine and metabolic problems in canine and cats.
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Extra resources for Canine and Feline Endocrinology, 4e
The modiﬁed water deprivation test alone is consistently correct in establishing a diagnosis of severe CDI, because direct measure of AVP concentrations does not alter the results of tests in which a patient did not concentrate urine during dehydration. However, inaccuracies may occur in differentiating patients with partial CDI from those with primary polydipsia and primary NDI; incorporating plasma AVP determinations into the modiﬁed water deprivation test helps differentiate these disorders.
MRI has been used to identify the presence of vasopressin in the posterior pituitary in humans. On T1-weighted images, MRI produces a bright spot in the sella caused by stored hormone in the neurosecretory granules in the posterior pituitary (Moses et al, 1992; Kurokawa et al, 1998). , 2003). The bright spot decreases with a prolonged stimulus for vasopressin secretion and has been variably reported in other polyuric disorders (Fujisawa et al, 2004). A bright spot in the region of the posterior pituitary has also been identified in dogs and presumably represents vasopressin stored in neurosecretory | CHAPTER 1 Water Metabolism and Diabetes Insipidus 29 BOX 1-5 T herapies Available for Polydipsic/Polyuric Dogs with Central Diabetes Insipidus, Nephrogenic Diabetes Insipidus, or Primary (Psychogenic) Polydipsia FIGURE 1-29 Magnetic resonance imaging (MRI) T1-weighted transverse image of the pituitary gland in a healthy adult dog illustrating the hyperintense “bright spot” (arrow) in the sella.
This seemingly paradoxic effect is seen in NDI, as well as in CDI, suggesting that this therapeutic agent has a mode of action distinct from that of chlorpropamide. By inhibiting sodium reabsorption in the ascending limb of the loop of Henle, the thiazides reduce total body sodium concentrations, thus contracting the ECF volume and increasing salt and water resorption in the proximal renal tubule. This results in lower sodium concentrations in the distal renal tubule and less osmotic effect to maintain tubular volume, resulting in a reduction in urine volume.